Cyhoeddiadau 2019

Mae'r cynnwys hwn ar gael yn Saesneg yn unig.

Al-Ansari, A. and Robertson, N. P. 2019. Cluster headache: an overview of established and emerging treatments. Journal of Neurology 266 (11), pp.2890-2892. (10.1007/s00415-019-09548-x)
Barker, R. A. et al., 2019. Designing stem-cell-based dopamine cell replacement trials for Parkinson's disease. Nature Medicine 25 (7), pp.1045-1053. (10.1038/s41591-019-0507-2)
Bernardi, R. E. et al., 2019. The inhibition of RasGRF2, but not RasGRF1, alters cocaine reward in mice. Journal of Neuroscience 39 (32), pp.6325-6338. (10.1523/JNEUROSCI.1120-18.2019)
Bhargava, R. , Daughters, K. L. and Rees, D. A. 2019. Oxytocin therapy in hypopituitarism: challenges and opportunities. Clinical Endocrinology 90 (2), pp.257-264. (10.1111/cen.13909)
Bourbon Teles, J. et al., 2019. Myelin breakdown in human Huntington's disease: multi-modal evidence from diffusion MRI and quantitative magnetization transfer. Neuroscience 403 , pp.79-92. (10.1016/j.neuroscience.2017.05.042)
Cavenagh, A. , Chatterjee, S. and Davies, W. 2019. Behavioural and psychiatric phenotypes in female carriers of genetic mutations associated with X-linked ichthyosis. PLoS ONE 14 (2) e0212330. (10.1371/journal.pone.0212330)
Crawford, K. et al. 2019. Medical consequences of pathogenic CNVs in adults: Analysis of the UK Biobank. Journal of Medical Genetics 56 , pp.131-138. (10.1136/jmedgenet-2018-105477)
Davies, W. 2019. An analysis of Cellular Communication Network (CCN) proteins as candidate mediators of postpartum psychosis risk. Frontiers in Psychiatry 10 876. (10.3389/fpsyt.2019.00876)
Davies, W. and Wilkinson, L. 2019. Editorial overview: special issue on epigenetics and genomic imprinting. Current Opinion in Behavioral Sciences 25 , pp.iii-v. (10.1016/j.cobeha.2018.12.006)
Drakesmith, M. et al. 2019. Genetic risk for schizophrenia and developmental delay is associated with shape and microstructure of midline white-matter structures. Translational Psychiatry 9 (1) 102. (10.1038/s41398-019-0440-7)
Drew, C. J. G. et al. 2019. A protocol for a randomised controlled, double-blind feasibility trial investigating fluoxetine treatment in improving memory and learning impairments in patients with mesial temporal lobe epilepsy: Fluoxetine, Learning and Memory in Epilepsy (FLAME trial). Pilot and Feasibility Studies 5 (1) 87. (10.1186/s40814-019-0474-x)
Dwyer, D. , Gasalla Canto, P. and Lopez, M. 2019. Partial reinforcement and conditioned taste aversion: No evidence for resistance to extinction. Quarterly Journal of Experimental Psychology 72 (2), pp.274-284. (10.1080/17470218.2017.1347191)
Egmond, M. E. et al., 2019. Variable interpretation of the dystonia consensus classification items compromises its solidity. Movement Disorders 34 (3), pp.317-320. (10.1002/mds.27627)
Fjodorova, M. et al. 2019. CTIP2-regulated reduction in PKA-dependent DARPP32 phosphorylation in human medium spiny neurons: implications for Huntington’s disease. Stem Cell Reports 13 (3-6), pp.448-457. (10.1016/j.stemcr.2019.07.015)
Föcking, M. et al., 2019. Complement pathway changes at age 12 are associated with psychotic experiences at age 18 in a longitudinal population-based study: evidence for a role of stress. Molecular Psychiatry 26 , pp.524-533. (10.1038/s41380-018-0306-z)
Fung, W. K. W. and Peall, K. J. 2019. What is the role of the cerebellum in the pathophysiology of dystonia?. Journal of Neurology 266 (6), pp.1549 -1551. (10.1007/s00415-019-09344-7)
Huckins, L. M. et al., 2019. Gene expression imputation across multiple brain regions provides insights into schizophrenia risk. Nature Genetics 51 , pp.659-674. (10.1038/s41588-019-0364-4)
Humby, T. and Davies, W. 2019. Brain gene expression in a novel mouse model of postpartum mood disorder. Translational Neuroscience 10 (1), pp.168-174. (10.1515/tnsci-2019-0030)
Kedia, G. et al., 2019. So Pretty! The neural correlates of self-other vs familiar-other attractiveness comparisons. Social Neuroscience 14 (1), pp.41-52. (10.1080/17470919.2017.1397544)
Kopczynska, M. et al. 2019. Complement system biomarkers in first episode psychosis. Schizophrenia Research 204 , pp.16-22. (10.1016/j.schres.2017.12.012)
Lancaster, T. M. et al. 2019. Structural and functional neuroimaging of polygenic risk for schizophrenia: a recall-by-genotype-based approach. Schizophrenia Bulletin 45 (2), pp.405-414. (10.1093/schbul/sby037)
Lansdown, A. et al. 2019. Regional cerebral activation accompanies sympathoexcitation in women with polycystic ovary syndrome. Journal of Clinical Endocrinology and Metabolism 104 (9), pp.3614-3623. (10.1210/jc.2019-00065)
Lipp, I. et al. 2019. Comparing MRI metrics to quantify white matter microstructural damage in multiple sclerosis. Human Brain Mapping 40 (10), pp.2917-2932. (10.1002/hbm.24568)
Manivannan, S. et al. 2019. The effectiveness of virtual reality interventions for improvement of neurocognitive performance post-traumatic brain injury: a systematic review. Journal of Head Trauma Rehabilitation 34 (2), pp.E52-E65. (10.1097/HTR.0000000000000412)
Matias, L. , Dwyer, D. and Gasalla Canto, P. 2019. Conditioned hedonic responses elicited by contextual cues paired with nausea or with internal pain. Behavioral Neuroscience 133 (1), pp.86-97. (10.1037/bne0000291)
Nadadhur, A. G. et al., 2019. Neuron-glia interactions increase neuronal phenotypes in tuberous sclerosis complex patient iPSC-derived models. Stem Cell Reports 12 (1), pp.42-56. (10.1016/j.stemcr.2018.11.019)
Niarchou, M. et al. 2019. Psychiatric disorders in children with 16p11.2 deletion and duplication. Translational Psychiatry (10.1038/s41398-018-0339-8)
Noakes, Z. et al. 2019. Human pluripotent stem cell-derived striatal interneurons: differentiation and maturation in vitro and in the rat brain. Stem Cell Reports 12 (2), pp.191-200. (10.1016/j.stemcr.2018.12.014)
Plumbly, W. et al. 2019. L-type voltage-gated calcium channel regulation of in vitro human cortical neuronal networks. Scientific Reports 9 13810. (10.1038/s41598-019-50226-9)
Ranson, A. et al. 2019. Top-down suppression of sensory cortex in an NMDAR hypofunction model of psychosis. Schizophrenia Bulletin 45 (6), pp.1349-1357. (10.1093/schbul/sby190)
Richards, A. et al. 2019. Associations between schizophrenia genetic risk, anxiety disorders and manic/hypomanic episode in a New Zealand longitudinal population cohort study. British Journal of Psychiatry 214 (2), pp.96-102. (10.1192/bjp.2018.227)
Salunkhe, G. et al., 2019. The impact of the COMT genotype and cognitive demands on facets of intra-subject variability. Brain and Cognition 132 , pp.72-79. (10.1016/j.bandc.2019.03.002)
Schwarz, E. et al., 2019. Reproducible grey matter patterns index a multivariate, global alteration of brain structure in schizophrenia and bipolar disorder. Translational Psychiatry 9 (1), pp.-. 12. (10.1038/s41398-018-0225-4)
Silva, A. I. et al. 2019. Reciprocal white matter changes associated with copy number variation at 15q11.2 BP1-BP2: A diffusion tensor imaging study. Biological Psychiatry 85 (7), pp.563-572. (10.1016/j.biopsych.2018.11.004)
Srinivasan, R. et al. 2019. Mental health and behavioural problems in children with XXYY: a comparison with intellectual disabilities. Journal of Intellectual Disability Research 63 (5), pp.477-488. (10.1111/jir.12607)
Sykes, L. et al. 2019. Genetic variation in the psychiatric risk gene CACNA1C modulates reversal learning across species. Schizophrenia Bulletin 45 (5), pp.1024-1032. (10.1093/schbul/sby146)
Toste, C. et al. 2019. No effect of genome-wide significant schizophrenia risk variation at the DRD2 locus on the allelic expression of DRD2 in post-mortem striatum. Molecular Neuropsychiatry 5 (4), pp.212-217. (10.1159/000501022)
Vasalauskaite, A. , Morgan, J. E. and Sengpiel, F. 2019. Plasticity in adult mouse visual cortex following optic nerve injury. Cerebral Cortex 29 (4), pp.1767-1777. (10.1093/cercor/bhy347)
Vorobyov, V. et al., 2019. Loss of midbrain dopamine neurons and altered apomorphine EEG effects in the 5xFAD mouse model of Alzheimer's disease. Journal of Alzheimer's Disease 70 (1), pp.241-256. (10.3233/JAD-181246)
Whale, R. et al., 2019. Factor analyses differentiate clinical phenotypes of idiopathic and interferon-alpha-induced depression.. Brain, Behavior, and Immunity 80 , pp.519-524. (10.1016/j.bbi.2019.04.035)